Three, nerve root injury (a) cause nerve root injury 1 extrusion injury of nerve roots and the degree of squeezing pressure, velocity, pressure range and the length of time and other factors. It caused only temporary nerve conduction disturbance, the weight can broken nerve fibers, caused by distal nerve degeneration. According to the compression factor is different, can be divided into exogenous and endogenous two. The former is squeezing factor in vitro injury, the latter is bruised body tissue. 2 traction injury in traffic accident. Such as centrifugal force body, cause nerve avulsion. Light person can pull off neural stem within the nerve bundle and vascular bundle, the neural stem internal bleeding, finally the formation of scar can be completely torn; orBroken neural stem or from the nerve root avulsion. 3 friction injury nerve bypass the process of bone, neural groove can cause chronic friction injury. Performance for the epineurium thickening or thinning of time can lead to nerve, scar formation. (two) the pathological changes of nerve root injury 1 damage response neural stem outer epineurium, in the outer membrane of nerve bundle surrounded by is, the number of different. Every nerve bundles by beam membrane. Every nerve includes many nerve fiber bundle, and nerve fibers and endoneurium surrounded by. Between the nerve membrane is composed of micro vascular network connections with. The physiological function of the perineurium of permeability barrier function, namely the blocking effect on the protein into play, to keep and maintain the physiological metabolism of tissue and fluid exchange. When nerve injury, nerve bundle membrane cannot maintain the barrier function of normal, exudate of albumin into, this is a kind of pathological infiltration, can cause inflammatory reaction, endoneurial edema, and then causes the nerve fiber damage, resulting in fibrosis and scar formation. Mast cell nerve sheath microvascular wall, in the perineurium injury, the release of endogenous inflammatory substances such as histamine, serotonin, 5-, is to stimulate the sensory nerve fiber algogenic substance. 2 damage to the microcirculation of the nerve root blood supply, remote from the spinal cord vascular, proximal from the intermediate segment artery, the two systems coincide in 1/3 nerve root, the Department of vascular network development is not sufficient, it is very easy to damage location. Interlinked nerve sheath of microvascular and external microcirculation. Microsurgical vascular nerve membrane, sympathetic nerve fibers innervating sympathetic chain, when stimulated, can cause microvascular contraction, or even to stop the flow of blood through blood. On one hand, the blood supply of nerve will change to a different degree, neural will also due to ischemia caused pathological changes, the beginning can cause paralysis or numbness, when the ischemic time with endoneurial microvascular hypoxic damage, albumin leakage increased, hematoma occurred in endoneurium, nerve function will not be restored. On the other hand, nerve injury, nerve bundle membrane increased microvascular permeability, which contains rich protein exudation increased and spread to the nerve gap, mast cells release inflammatory vascular injury of chemical medium, the medium is inflammation, pain caused by substances. Thus clinically with pain as the main feature. This means that any improvement in the microcirculation of nerve root, can make the pain remission. 3 nerve root compression of nerve root is relatively well protected, not easily affected by the surrounding tissue and traumatic injury. But because it has not the nerve root and peripheral nerve that connective tissue sheath, so the direct mechanical compression caused by spinal diseases especially sensitive. Nerve root compression, first occurrence of ischemia and hypoxia, ischemia, hypoxia, and the effect on nerve conduction function more than the stress itself. At the same time, the local nerve compression, direct mechanical effect on nerve, including nerve fiber deformation, node of Ranvier node Zhou Suiqiao shift and stripping, even low levels of compression, 4kPa pressure can cause the change of axial flow, protein from nerve cells to the distal end of the axon transport damage, 26.7kPa compression, caused by change, is secondary to neural nutrition supply damage. Nerve root pressure and stimulation, cause inflammation, exudation, edema, hyperplasia, more heavy pressure on the nerve root. 4 nerve root inflammation is mainly due to intervertebral disc rupture of annulus and nucleus pulposus protrusion, oppression and stimulate the nerve root, caused by traumatic inflammation. At the same time, fibrous ring rupture and nerve root may cause the inflammatory edema, aggravate nerve compression, the outer anular dominant sinus vertebral nerve, stimulation of this nerve can cause painful waist, hips. Inflammation of the sciatic nerve root pain fibers, short circuit, causing severe low back pain and sciatica. For a long time, nerve root adhesion and -- fibrosis and rupture, projection, the sensory and motor dysfunction of persistence. 5 nerve fibers and nerve fibers after concussion concussion, no significant changes in the organizational structure, electric response still exists, but there may be a temporary loss of conduction function. Such as the damage caused by the destruction of nerve fiber structure, electric reaction disappeared, conduction loss of function.......
The first chapter of lumbar and around the applied anatomy of the
lumbar and connected to the
second section connected lumbar vertebral canal and its contents.
two spinal canal, spinal canal and the contents of
third lumbosacral muscle
two spine extensors, lumbar spine, lumbar spine
three flexor flexor muscle of
four, lumbar spine rotators
Fourth lumbosacral vessels.
A, lumbosacral artery
three lumbosacral vein, the blood supply of the intervertebral disc.
a nerve, lumbar, sacral nerve
three lumbosacral sympathetic
sixth lumbar, sacral bony landmarks and the surrounding structure of
A, lumbar osteoarthritis sign < br / > two, lumbosacral region
three bony landmarks, lumbosacral area bone and joint
seventh lumbar protrusion of the intervertebral disc.
A, the anatomical basis of prolapse of lumbar intervertebral disc degeneration of the anatomical basis for
two, prolapse of lumbar intervertebral disc pathological types of anatomical base Basic
three, lumbar curvature abnormality and the clinical significance of
second chapter biomechanical
the first day the biomechanics of several basic concepts of
a > two
three, elastic and elastic modulus of
Second viscoelastic characteristics of lumbar vertebrae and spinal physiological and the mechanical properties of
A, biomechanical functions of the spine.
two, spinal biomechanics and anatomy of
three, the function of spine unit
four, the mechanical properties of
five spine, intervertebral disc biomechanical
six, vertebral arch and facet joint mechanical properties of
seven, ligament mechanics characteristics of
third lumbar vertebrae and spinal physiological curvature and biomechanical
A, spinal physiological curvature of the formation of
two, affect the spinal physiological curvature factor
three, spinal physiological curvature biomechanical
fourth lumbar kinematic
A, lumbar spinal motion and mechanics analysis
two, lumbar movement characteristics of
three, lumbar range of motion
Four, lumbar movement on the spinal canal contents of
fifth lumbar stability and instability of
A, the stability of the spine and spinal instability concept
two, relationship between structure and stability of lumbar
three, lumbar muscle of spine stabilization effect of < br > four, lumbar structure plasticity
five, clinical instability of the lumbar spine anatomy and biomechanical factors
six, function training on lumbar stability effect of
sixth spinal statics and dynamics of
A, spinal mechanics in general < br > two, different postures and movements of lumbar spine stress
third chapter of prolapse of lumbar intervertebral disc the cause of disease pathology in
fourth chapter TCM etiology and pathogenesis of
fifth chapter of prolapse of lumbar intervertebral disc check
sixth chapter of lumbar disc herniation in diagnosis and differential diagnosis of
seventh chapter of lumbar disc herniation syndrome differentiation of
eighth chapter of lumbar disc herniation Chinese medicine and Western medicine combined with treatment of
the ninth chapter of lumbar disc herniation and Western medicine
tenth chapter of lumbar disc herniation complications and treatment of
eleventh chapter of lumbar intervertebral disc protrusion rehabilitation therapy.
twelfth chapter of prolapse of lumbar intervertebral disc @##@ prevention The book consists of 12 chapters, system, detailed, in-depth discussion of the protrusion of the lumbar intervertebral disc disease, including the applied anatomy of lumbar disc herniation and biomechanics, lumbar disc herniation, pathogenesis, etiology and pathology examination, diagnosis and differential diagnosis, TCM, treatment combined with traditional Chinese medicine, western medicine and integrated Chinese and Western medicine, rehabilitation and prevention. Emphatically introduces all kinds of methods of treatment of lumbar disc herniation in traditional Chinese medicine, western medicine and integrated traditional Chinese and Western medicine and its progress, such as the small needle knife therapy, Chinese medicine iontophoresis, epidural drug perfusion, interventional therapy, minimally invasive operation etc.. This book is a summary of the Department of orthopedics experts experience combined with traditional Chinese medicine, western medicine and integrated traditional Chinese and Western medicine. With practical content, readable. Mainly used in orthopedics and traumatology, scientific research professionals can also be used for reference, for prolapse of lumbar intervertebral disc disease study.
"Published by people's Medical Publishing House in the treatment of lumbar disc herniation with combination of Chinese traditional and Western medicine".
Medical Science @ 2017